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NMN AND NAD+
THE KEY TO STAY YOUNG AND HEALTHY

Nicotinamide mononucleotide (NMN), a key NAD+ intermediate, has been shown to enhance NAD+ biosynthesis and ameliorate various pathologies in research.

After 12-month-long study, Orally administered NMN was quickly utilized to synthesize NAD+ in tissues. Remarkably, NMN effectively mitigates age-associated physiological decline.

Without any obvious toxicity or deleterious effects, NMN suppressed age-associated body weight gain, enhanced energy metabolism, promoted physical activity, improved insulin sensitivity and plasma lipid profile, and ameliorated eye function and other pathophysiologies.

Consistent with these phenotypes, NMN prevented age-associated gene expression changes in key metabolic organs and enhanced mitochondrial oxidative metabolism and mitonuclear protein imbalance in skeletal muscle. These effects of NMN highlight the preventive and therapeutic potential of NAD+ intermediates as effective anti-aging interventions in humans.

ENERGY-LEVEL

MAINTAINS ENERGY LEVELS

general-healty

SUPPORTS WELLBEING AND CIRCULATION

FREE-RADICALS

REDUCES FREE RADICALS

man-health-icon

SUPPORT MALE SEXUAL FUNCTION

general-healty

MAINTAINS GENERAL HEALTH

herbal-base

CONTAINS TRADITIONAL HERBAL MEDICINES

NEW MEDIA RELEASES ABOUT THE EZZ AND NMN

Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice

Mills et al. conducted a 12-month-long administration of nicotinamide mononucleotide (NMN)

Loss of NAD Homeostasis Leads to Progressive and Reversible Degeneration of Skeletal Muscle

NAD levels decline in multiple tissues with age or in disease. Frederick et al. show that impaired intramuscular NAD synthesis compromises skeletal muscle mass…

NAD+ Replenishment Improves Lifespan and Healthspan in Ataxia Telangiectasia Models via Mitophagy and DNA Repair

Mitochondrial damage and NAD+ depletion are key features in ataxia telangiectasia.

NMN & NAD+ TARGETTING MANY HEALTH AND WELLBEING RELATED ISSUES